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An impairment of these mechanisms underlying UV-B–induced immunosuppression may be important in the pathogenesis of PMLE.The study found similar immunosuppression of contact sensitization to diphenylcyclopropenone due to earlier exposure to solar-simulating UV radiation between both groups.The prevalence of PMLE was 32 (0.65%) in 4899 residents and was 3.8 times higher in women compared with men. notes that PMLE is the most common photodermatitis after chronic actinic dermatitis, while hydroa vacciniforme and solar urticaria are uncommon.At higher elevations, the prevalence of PMLE increased. Furthermore, this report stated lichenoid PMLE and pin-point papular PMLE are the most common types found in the subcontinent.The possibility that a subset of PMLE called benign summer light eruption or BSLE which might be milder and might be more UVA driven has been suggested by an Italian group.

The reduction of these cytokines seems linked to a relative neutropenia and is a manifestation of decreased Langerhans cell migration and reduced TH2 skewing.The psychological impact was related to the predicted consequences of PMLE, whereas health-related variables played a lesser role. Courtesy of Waikato District Health Board and Derm Net New Zealand ( Wz Y0MCw0ODBd/Watermarked Wy Iy NTg0MCJd/pmle-22. The etiology of polymorphous light eruption (PMLE) is not fully known, and it is likely to be multifactorial.Women associated more severe consequences linked to PMLE and were more emotionally distressed than men. The immunologic pathogenesis of PMLE is supported by the study of timed biopsy samples of PMLE lesions.PMLE is characterized by recurrent, abnormal, delayed reactions to sunlight, ranging from erythematous papules, papulovesicles, and plaques to erythema multiforme –like lesions on sunlight-exposed surfaces.Within any 1 patient, only 1 clinical form is consistently manifested.

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